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Yale University
School of Medicine
Department of Neurology
P.O. Box 208018
New Haven, CT
06520-8018
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Albert Lo, MD, PhD
Assistant Professor

Department of Neurology
Yale University School of Medicine
P.O. Box 208018
New Haven, CT 06520-8018
Office: 203.932.5711 ext 5734
Lab: 203.937.3802
Fax: 203.937.3801
email: albert.lo@yale.edu
Training:
1998-2001: Residency training, Yale Neurology Residency Program Yale University
School of Medicine, New Haven, CT
1997-1998: Internship, Yale Primary Care Internal Medicine Program, Yale
University School of Medicine, New Haven, CT
1991-1997: MD, PhD, Wake Forest University School of Medicine, Winston-Salem,
NC
1990-1991, CPH, Harvard School of Public Health, Cambridge, Massachusetts
1982-1987, BA, University of California Berkeley, Berkeley, California
Expertise:
Multiple Sclerosis, Neurorehabilitation
Research Interests:
Pathobiology of demyelinating disease, neuronal injury and repair, neuroprotection,
MS epidemiology, robotic neurorehabilitation, clinical trial design and
methodology
Selected Publications: Lo AC, Hadjimichael, Vollmer TL (2004).
Treatment patterns of multiple sclerosis patients: A comparison of veterans
and non-veterans using the NARCOMS registry. (Submitted for publication).
Hains BC, Saab CY, Lo AC, Black JA, Waxman SG. Stabilization of Sodium
Influx with Phenytoin Enhances outcome Recovery Following Contusion. (In
Press, 2004).
Craner MJ, Hains BC, Lo AC, Black JA, Waxman SG (2004). Co-localization
of sodium channel Na 1.6 and the sodium-calcium exchanger at sites of
axonal injury in the spinal cord in EAE. Brain. 127: 294-303
Lo AC, Saab CY, Black JA, and Waxman SG (2003). Phenytoin protects spinal
cord axons and preserves axonal conduction and neurological function in
a model of neuroinflammation in vivo. J. Neurophysiol. 90: 3566-3571
Craner MJ, Lo AC, Rasband MN, Black JA, and Waxman SG (2003). Abnormal
sodium channel distribution in the optic nerve in a model of inflammatory
demyelination. Brain 126: 1552-1561.
Lo AC, Black JA, Waxman SG (2002). Neuroprotection of axons with phenytoin
in experimental allerigic encephalomyelitis. Neuroreport 13:1909-1912.
Gould TW, Burek MJ, Ishihara R, Lo AC, Prevette D, and Oppenheim RW. (1999).
Androgens rescue avian embryonic lumbar spinal motoneurons from injury-induced
but not naturally occurring cell death. J. Neurobiol. 41:585-95.
Lo AC, Li L, Oppenheim RW. Prevette D and Houenou LJ. (1995). Ciliary
neurotrophic factor (CNTF) promotes survival of spinal sensory neurons
following axotomy but not during the period of programmed cell death.
Exp. Neurol. 134:49-55.

    
 

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