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Yale University
School of Medicine
Department of Neurology
P.O. Box 208018
New Haven, CT
06520-8018

Albert Lo, MD, PhD
Assistant Professor

Department of Neurology
Yale University School of Medicine
P.O. Box 208018
New Haven, CT 06520-8018

Office: 203.932.5711 ext 5734
Lab: 203.937.3802
Fax: 203.937.3801

email: albert.lo@yale.edu

Training:

1998-2001: Residency training, Yale Neurology Residency Program Yale University School of Medicine, New Haven, CT
1997-1998: Internship, Yale Primary Care Internal Medicine Program, Yale University School of Medicine, New Haven, CT
1991-1997: MD, PhD, Wake Forest University School of Medicine, Winston-Salem, NC
1990-1991, CPH, Harvard School of Public Health, Cambridge, Massachusetts
1982-1987, BA, University of California Berkeley, Berkeley, California

Expertise:
Multiple Sclerosis, Neurorehabilitation

Research Interests:
Pathobiology of demyelinating disease, neuronal injury and repair, neuroprotection, MS epidemiology, robotic neurorehabilitation, clinical trial design and methodology

Selected Publications:
Lo AC, Hadjimichael, Vollmer TL (2004). Treatment patterns of multiple sclerosis patients: A comparison of veterans and non-veterans using the NARCOMS registry. (Submitted for publication).
Hains BC, Saab CY, Lo AC, Black JA, Waxman SG. Stabilization of Sodium Influx with Phenytoin Enhances outcome Recovery Following Contusion. (In Press, 2004).
Craner MJ, Hains BC, Lo AC, Black JA, Waxman SG (2004). Co-localization of sodium channel Na 1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE. Brain. 127: 294-303
Lo AC, Saab CY, Black JA, and Waxman SG (2003). Phenytoin protects spinal cord axons and preserves axonal conduction and neurological function in a model of neuroinflammation in vivo. J. Neurophysiol. 90: 3566-3571
Craner MJ, Lo AC, Rasband MN, Black JA, and Waxman SG (2003). Abnormal sodium channel distribution in the optic nerve in a model of inflammatory demyelination. Brain 126: 1552-1561.
Lo AC, Black JA, Waxman SG (2002). Neuroprotection of axons with phenytoin in experimental allerigic encephalomyelitis. Neuroreport 13:1909-1912.
Gould TW, Burek MJ, Ishihara R, Lo AC, Prevette D, and Oppenheim RW. (1999). Androgens rescue avian embryonic lumbar spinal motoneurons from injury-induced but not naturally occurring cell death. J. Neurobiol. 41:585-95.
Lo AC, Li L, Oppenheim RW. Prevette D and Houenou LJ. (1995). Ciliary neurotrophic factor (CNTF) promotes survival of spinal sensory neurons following axotomy but not during the period of programmed cell death. Exp. Neurol. 134:49-55.



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